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Forced expression of the above-mentioned miRNAs affected tumorigenic properties, (i.e

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The peptide fragment has also been suggested to promote GH secretion by regulating physiological processes involved in GH synthesis

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Therefore, SCFAs deficiency may represent a contributing factor to PCOS symptomatology, potentially through multiple interconnected mechanisms including exacerbating ovarian inflammation, impairing follicular maturation, aggravating IR, and promoting follicular atresia via direct suppression of glucose uptake in granulosa cells

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