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The functions of glutathione peroxidase in ROS homeostasis and fruiting body development in Hypsizygus marmoreus

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Why GLP-1s are Biologically Plausible for Alzheimers Beyond lowering glucose and weight, GLP-1 signaling appears to: Improve insulin signaling in the brain Dampen neuroinflammation (microglial activation) Reduce oxidative stress Interact with amyloid- and tau pathways in preclinical models Recent peer-reviewed reviews synthesize these mechanisms and frame GLP-1s as potential multi-target neuroprotective agents rather than single-pathway drugs

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Notably, GLP-1R-LepR co-expressing cells proved to be crucial targets of newly synthesized GLP-1/leptin dual agonists, which create a negative energy balance and assist the action of leptin [75]