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362 Considering the expression of the GLP-1R in thyroid C cells and the ability of GLP-1 to stimulate calcitonin secretion through a cAMP-mediated mechanism in vitro, it is plausible that calcitonin plays a role in the alterations in bone metabolism observed in GLP-1R-treated animals

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These processes induce microvascular constriction, blood cell aggregation, cerebral edema, and neuronal apoptosis, synergistically exacerbating pathological cascades (Zhang et al., 2024), and impeding clinical management with compromised prognostic accuracy

Collagen formation, on the other hand, naturally declines with age or exposure to environmental stressors

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