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Bypass First-Pass Metabolism: Improves systemic bioavailability and potency
Apoptosis in arsenic trioxide-treated Calu-6 lung cells is correlated with the depletion of GSH levels rather than the changes of ROS levels
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Linagliptin but not sitagliptin inhibited transforming growth factor-beta2-induced endothelial DPP-4 activity and the endothelial-mesenchymal transition
It has several defence mechanisms to evade oxidative and nitrosative agents released by phagocytic host cells during infection
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