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Mechanism of Action Acetyl Group Protection : The attached acetyl group acts as a molecular shield, preventing digestive enzymes from breaking down glutathione prematurely in the stomach and intestines, which allows the intact molecule to reach target tissues like the liver, lungs, and brain
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Exposure to ototoxic drugs, such as aminoglycoside antibiotics (such as streptomycin, gentamicin, acamicin, etc.), non-steroidal anti-inflammatory drugs, loop diuretics, antineoplastic drugs (such as cisplatin, nitrogen mustard, etc.), or inhalation of harmful gases (carbon monoxide, hydrogen sulfide, etc.), all can cause ARHL
Stimulation of TNFR1 induces certain conformational changes in the receptor, enabling its cytosolic portion to recruit several proteins to form the prosurvival complex I consisting of TNFR-associated death domain (TRADD), TNFR-associated factor 2 and 5 (TRAF2/5), RIPK1, cellular inhibitor of apoptosis proteins 1/2 (cIAP1/2), and linear ubiquitin chain assembly complex (LUBAC) (Nikoletopoulou et al., 2013
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