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Although current research indicates that ferroptosis plays a central regulatory role in the pathological process of FRDAlinking mitochondrial iron accumulation, neurodegeneration, and cardiomyopathyand provides a new theoretical anchor for understanding the FXN deficiencyiron dysregulationtarget-organ injury axis, several fundamental limitations remain

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In wound healing studies, GHK-Cu injected into one area of the body (such as thigh muscles) improved healing at completely different locations (such as the ears)

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