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Dziaaniu przeciwzapalnemu redukuje stres oksydacyjny oraz moduluj aktywno cytokin prozapalnych (TNF-, IL-6)
Huguet et al, 2016)
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These clinical and molecular findings were paralleled by in vitro wound-healing assays, in which HA + GSH-C4 significantly accelerated epithelial repair under basal, inflammatory, and oxidative conditions, further supporting its protective and reparative potential
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