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[6] NAD+ augmentation may counteract age-related NAD+ loss caused by overactive NAD-consuming enzymes (such as the NADase CD38), thereby supporting mitochondrial maintenance and metabolic homeostasis
Pinto, D., Robine, S., Jaisser, F., El Marjou, F
In pre-clinical research, it is primarily studied for its effects on gastrointestinal healing, tendon and ligament repair, vascular growth (angiogenesis), and potential neuroprotective properties
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As the causative bacteria die off due to antibiotics, inflammatory agents are released and temporarily worsen symptoms before improvement is noted
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