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Carnitine depletion occurs rapidly during prolonged ischemia (Carvajal and Moreno-Snchez 2003)
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At the initial baseline visit, serial venous sampling performed after oral d 3 -l-carnitine challenge revealed rapid increases in plasma concentrations of d 3 -l-carnitine, and subsequent increases in d 3 -BB and d 3 -TMAO following a lag phase (Figure 1, AC, filled circles)
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Of note, necroptosis of erythrocytes was associated with the formation of the RIPK1/ FADD/caspase-8 complex, and caspase-8 activation prevented the occurrence of necroptosis
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