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4, eight trials totaling 13655 patients were included with Semaglutide as the experimental group

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Consequently, impaired PPAR signaling disrupts mitochondrial fatty acid -oxidation, leading to lipid accumulation in hepatocytes and possibly accelerating the progression of MASLD [297]

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Oxidative stress and SASP are revealed as deeply interconnected processes, emphasizing the need for dual-targeting approaches that restore redox balance while modulating the senescent cell secretome