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Elevated plasma carnitine levels likely inhibit endogenous production by suppressing -butyrobetaine hydroxylase, the enzyme which converts BB to carnitine (5), therefore leading to an accumulation of BB

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Conclusions/interpretation Triagonist-induced augmentation of GSIS is primarily mediated through its interaction with the GLP-1 receptor and subsequent activation of the GqTRPM5 signalling pathway

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