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PARP1 is activated by DNA damage resulting in the depletion of cellular NAD + pools that hampers cellular energy metabolism by reducing SIRT1 activity [33, 34]

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Glyoxalase I was found to be selectively overexpressed in antiapoptotic UK711 cells [87]

All interventions were compared with current use of SGLT2is (20% of the total population) and GLP-1 RAs (5% of the total population)

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