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Because it circulates systemically, it can act on every active injury site at once
NAC treatment, however, caused only moderate recovery of aspirin-induced apoptosis in HepG2 cells suggesting additional factor(s), other than altered GSH pool, which could play a role in inducing apoptosis by ASA
Interestingly, sex-specific differences emerged: females exhibited more pronounced downregulation of metabolic pathways, while males showed stronger immune activation
The impact on TMAO levels and whether this causally increases CVD risk is still uncertain
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