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This activation triggers a cascade of downstream signaling pathways, including the phosphorylation of effectors such as 4EBP1 and S6K1, which regulate protein and lipid synthesis

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It was demonstrated, that metabolic disturbances in hypothalamic astrocytes, caused either by selective knock-out of insulin receptors or decreased generation of reactive oxygen species (ROS) modulated glucose transport via BBB and also led to modification in behavioral responses in experimental animals [95, 96]

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