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Pagliaro LC, Liu B, Munker R, Andreeff M , Freireich EJ, Scheinberg DA, Rosenblum MG

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75 Conversely, forced overexpression of CDO1 in breast cancer cells shifted the flux from glutathione synthesis towards cysteine catabolism, consequently increasing ROS levels and leading to reduced cell viability and growth

The consequent impairment in oxalate homeostasis can trigger localized and systemic inflammation, progressive kidney disease and cardiovascular complications, including sudden cardiac death

The primary endpoint is percent change in body weight from baseline