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Mechanistically, rapamycin primarily inhibits mammalian target of rapamycin complex 1 (mTORC1), although chronic administration also attenuates mTORC2 signaling, while potently blocking the translation of membrane-bound IL-1

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Oxidized LDL (oxLDL) measures the amount of low-density lipoprotein cholesterol that has undergone oxidative modification

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This is due to the fact that GHK is able to inactivate the damaging byproducts released from free radical reactions