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High antigen levels are the cause of T cell exhaustion during chronic viral infection

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Abstract Reactive oxygen species (ROS), inevitable byproducts of aerobic respiration, exhibit both signaling and cytotoxic effects depending on concentration within cells

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Furthermore, the PfGrx-catalyzed oxidation of roGFP2 is more than four orders of magnitude faster than the uncatalyzed reaction with GSSG and occurs two orders of magnitude faster via the glutathione-dependent monothiol mechanism than by the glutathione-independent dithiol mechanism (Fig