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At baseline, subcutaneous white-adipose-tissue samples from the high-apoB group secreted markedly more interleukin-1beta (IL-1beta) in response to LDL and other stimuli than tissue from the healthy-apoB group

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This makes sense because both drugs act through GLP-1 receptor pathways, and GI effects are inherent to that mechanism

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3.5 Semaglutide intervention and fecal microbiota transplantation improved the dysbiosis of gut microbiota in obese mice, increasing the relative abundance of beneficial bacteria, and affecting the levels of short-chain fatty acids The species accumulation curve and dilution curve indicated that the sample size and sequencing depth were sufficient (Supplementary Figure S3A)

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