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Effective GLP-1 tracking requires monitoring both primary therapeutic outcomes and safety parameters
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possibilities include 1) increasing insulin sensitivity via phosphorylation of insulin receptors, which down-regulates insulin/IGF-1 signaling, 2) down-regulating the expression of inflammatory cytokines that inhibit NF-B activation, 3) directly activating the AMPK pathway to activate the p53 tumor suppressor gene, and 4) promoting cancer cell apoptosis via peroxisome proliferator-activated receptor gamma (PPAR) activation and inhibiting angiogenesis [58, 59]
Curr Opin Biotechnol 32:186194
Rahman, I., Bel, A., Mulier, B., Donaldson, K
The GIP component seems to buffer some of the GI effects of GLP 1 activation
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