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Molecular chaperones and protein folding as therapeutic targets in Parkinsons disease and other synucleinopathies

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Here, we employed two different experimental settings in which glutathione and NADP redox responses are diametrically opposed, to test whether NERNST responds predominantly to NADP or glutathione redox changes

BPC-157 promotes tendon-to-bone integration and accelerates the structural healing that tendons need to fully recover

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Hrd1-mediated ACLY ubiquitination alleviate NAFLD in db/db mice