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These data collectively suggest that the inhibitory effect of GSSG on the NF-B signaling pathway, via suppression of IKK- activation, is mechanistically implicated in the increased sensitivity of hepatocytes to TNF killing
& Prusoff, W
Due to (temporarily) reduced sodium reuptake while continuously promoting glucosuria, these drugs induce a reduction in GFR likely via activation of tubuloglomerular feedback (TGF) that may represent reduced intraglomerular pressure [5]
In addition, mitochondria are also the primary producers of intracellular reactive oxygen species (ROS), most of which originate from the mitochondrial respiratory chain [55]
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