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Such investigations are crucial not only for understanding tumor cells themselves but also for elucidating their significant effects on the function and fate of neighboring immune cell populations in the TME

Glossary Apoptosis A caspase-dependent programmed cell death pathway controlled by the BCL-2 protein family and mitochondrial factors that maintains tissue homeostasis and eliminates damaged or unwanted cells in a controlled manner by orchestrating distinct cellular changes, including chromatin condensation, DNA fragmentation and membrane blebbing

Crucially, FOXO4-DRI selectively induced apoptosis in senescent TM3 Leydig cells by causing nuclear exclusion of active Ser15-phospho-p53, leading to a significant increase in apoptosis rate and altering pro-apoptotic (BAX, cleaved-Caspase3 increase) and anti-apoptotic (BCL-2 decrease) protein levels

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Anti-aging and cellular damage repair slows down free radical damage and protects brain function