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Development of a cysteine-deprived and C-terminally truncated GLP-1 receptor
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[1] [2] It has not yet received regulatory approval from the Medicines and Healthcare products Regulatory Agency (MHRA) or the European Medicines Agency (EMA) at the time of writing
CCK exerts its biological actions by binding to specific G-protein coupled receptors (GPCR)
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1b), suggesting enhanced redox adaptive capacity in this group
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