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The peptide was observed to have the potential to prevent changes in monoamine oxidase type A (MAO-A) and serotonin levels

GHK (endogenous glycyl-L-histidyl-L-lysine) GHK-Cu (Copper Peptide) References: 1

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Given that EtOH-induced mitochondrial dysfunction and ER stress are intimately involved in the pathogenesis of ALD, future studies to examine these pathways under the current experimental setting will provide important mechanistic information underlying the protection seen in GCLM KO mice

This sleep phase may be understood as serving a restorative and therapeutic role in the central nervous system