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These agents enhance glucose-dependent insulin secretion from pancreatic beta cells, suppress inappropriate glucagon release, slow gastric emptying, and act on central nervous system appetite centers to promote satiety

Ultimately, understanding how GLP-1 therapies fit into the broader diabetes treatment algorithm helps improve outcomes and quality of life for patients

This analysis confirmed a significant association between GLP-1 RAs and these NAEs (ROR 1.44, 95% CI 1.411.46), with variation observed across different GLP-1 RAs (Fig

Patients should evaluate several important factors before starting treatment

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