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The antioxidant completely gets absorbed much more quickly when directly injected into the bloodstream [6] as opposed to when it gets administered orally
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Two possibilities are that DHA concentrations are extremely low in compartments such as the chloroplast and cytosol or that rate constants for the chemical reaction are much slower in the crowded cellular environment than those measured in dilute solutions in vitro, making enzymes necessary to favour the reaction between DHA and GSH
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SIRT3 mediates hippocampal synaptic adaptations to intermittent fasting and ameliorates deficits in APP mutant mice
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