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In summary, the kinetic data for the E170X mutants support a glutaredoxin-catalysed formation of GSSEtOH as well as an exclusive interaction between Glu170 and the glutathione moiety of GSSR in accordance with the glutathione-scaffold model
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And a significantly reduced ATP level was found in both 8-week and 10-week ethanol-exposed WT mice and APP/PS1 mice (Fig
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