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Our study revealed a novel role for GLP-1 and its derivatives in pain relief, suggesting exendin 2029 as a promising therapeutic candidate
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In IELs specifically, the activation of GLP-1R signaling suppresses phosphorylation of chainassociated protein kinase 70 (ZAP70) and its downstream target SH2 domaincontaining leukocyte protein 76 (SLP-76), leading to dampening of proximal T cell receptor signaling and reduced cytokine transcription
As a result, the excess free radicals begin to damage the bodys cells and tissues
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