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Less frequent AEs like hypoglycemia, is mostly seen in combination with insulin or sulfonylureas (157) and some reports of pancreatobiliary complications are partially explained by direct altered absorption of bile acids, rapid weight loss or changes in the gallbladder motility with increased risk of pancreatitis, even though it is extremely rare (160)

That's how we ended up with an injectable that lasts a full week in the body and can even reach receptors in the brain to help reduce appetite

You would need complications like severe heart failure, respiratory failure, or organ damage directly attributable to obesity

A meta-analysis of 24 weight-loss intervention studies (N=2,284) found that a 10 kg reduction in body weight raised total testosterone by approximately 2.9 nmol/L and SHBG by 5.7 nmol/L in men with obesity [3]

A recent mechanistic study using LDL receptordeficient mice and ox-LDLchallenged human endothelial cells demonstrated that liraglutide, one of the most widely studied GLP-1RAs, directly improves endothelial function by suppressing oxidative and inflammatory pathways ( in vitro experiments showed that liraglutide mitigated oxLDL-induced endothelial injury by suppressing NOX4 and NF-B signaling, and downregulating ICAM-1 and VCAM-1 expression (14)