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Multiple studies have demonstrated cardiac-specific benefits: Improved left ventricular function in GH-deficient models (Bisi et al., 1999) Anti-fibrotic effects on cardiac tissue Protection against ischemia-reperfusion injury in isolated heart models (Locatelli et al., 1999) Reduced cardiomyocyte apoptosis following ischemic damage These cardiac effects appear to be mediated through CD36 receptor activation independent of GH release making Hexarelin unique among GHRPs
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