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doi:10.2217/ijh-2020-0010 92

This distinction matters because exogenous GH administration carries a different risk profile than peptide-stimulated endogenous release

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These microbial compounds stimulate the Nrf2/Keap1 pathway by electrophilically modifying Keap1 cysteine residues or by increasing Nrf2 phosphorylation through the PI3K/Akt, ERK, and PKC signaling cascades [151]