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The researchers proposed that the compounds effects on muscle function operate through boosting NAD+ levels by inhibiting NNMTs consumption of nicotinamide, more substrate is available for NAD+ synthesis via the salvage pathway, supporting mitochondrial function and energy metabolism in aged skeletal muscle

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However, when amounts of fat exceed the adipose tissues storage capacity and overwhelm adipose tissues functional capacity, fat begins to deposit ectopically in other metabolically relevant organs, such as the liver, skeletal muscle, kidney, and pancreas

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