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Stern RS, Zierler S, Parrish JA

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Deficiencies in CPTII may result in exercise induced muscle injury due to inability to increase FA oxidation with increased exertion

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Although this study did not measure the response of neuronal AR deficiency on skeletal muscle phenotypic or functional outcomes, the data supported a role in neuronal AR for mitigating an obesogenic phenotype in an age-dependent manner

Published in Nature , this finding transformed the vocabulary of the field: compounds once called GHRP-like agonists were now understood as ghrelin mimetics or ghrelin receptor agonists, and the receptor gained an endogenous physiological anchor